Cerebral Palsy

Cerebral palsy is manifested by a syndrome of motor disorders, impaired muscle tone, postural maintenance, and slowed or absent motor development.

For a long time, the causes of cerebral palsy were considered to be intrauterine or perinatal brain damage caused by birth trauma, intracranial hemorrhage, ischemia, intrauterine infection, or brain malformation.

The clinical picture of CP was the development of spastic hemi- or tetraparesis of skeletal muscles, that is, increased tone in two or all four limbs.

Motor disorders are often combined with mental and speech developmental delay.

There is no specific treatment for cerebral palsy; the emphasis is on motor development through massage, gymnastics, physiotherapy, reflexology, hydrotherapy, swimming, and other methods.

In cases of high muscle tone, drugs to reduce tone and block the neuromuscular synapse may be used – botulinum toxin, tizanidine, tolperisone. Sometimes surgeries to transect conductive nerve pathways and tendon release are offered.

The very definition of CP does not contain an indication of the cause of motor disorders; it merely states the fact of these disorders. However, currently the etiology of motor disorders is much broader; the main cause is often mitochondrial pathology, that is, a disorder of metabolic processes directly in the muscles, as a result of which muscle cells synthesize critically little energy. This leads to muscle weakness and delayed motor development in the child. In such cases, exercise, massage, and gymnastics with such children do not give the desired effect and do not contribute to muscle strengthening.

Children with motor delays are often found to have concomitant somatic pathology, which is mistakenly attributed to neurological disorders. For example, drooling due to inflammation of the salivary glands is mistakenly attributed to neurological pseudobulbar syndrome, constipation to reduced motor activity, and protective stress hypertonicity to cerebral damage.

Standard neurological and orthopedic programs are often a great stress for the child, are painful, and worst of all, these sufferings are inflicted in the presence of parents; accordingly, the child-mother bond is disrupted and overall the rehabilitation process is slowed down.

The basis of rehabilitation for a child with motor disorders in our clinic is determining the true cause of the motor disorders, studying the child's metabolic status, examining their amino acid and micronutrient balance, and correcting them.

Basic mitochondrial support includes correction of function and reduction of intoxication levels, correction of constipation, and reduction of inflammation. For this purpose, necessary laboratory tests are conducted. At the clinic, according to indications, the KETOMIT program is introduced, the diet is adjusted, and poorly digestible foods and those contributing to intoxication are removed.

To increase the energy output of muscle cells, courses of mitochondrial support are carried out, including ozone therapy and interval hypoxic therapy. The clinic organizes a rehabilitation course REMIT, during which the patient significantly improves their motor status.